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WIN
ROUNDS |
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Welcome
to WIN ROUNDS! The objective of Windsor University School
of Medicine in introducing WIN ROUNDS is to make sure
that every Windsor student has knowledge and skills
in basic history taking, physical diagnosis, differential
diagnosis and management of the patient. Our students
enter into clinical sciences after completing their
basic medical sciences. As part of the clinical sciences
every student of Windsor is required to complete CORE
and ELECTIVE rotations in the hospital setting. |
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During
their clinical rotations our students rotate with Attending
Physicians in several hospitals. Our students see cases
in in-patient, out-patient, Emergency Medicine ( ER
), Intensive Care Unit, Operating Theatre, Satellite
centers, Nursing Homes and many other locations. During
their rotations on a daily basis while they are rotating
with their respective attending physicians, our students
are challenged with questions on all aspects of the
History taking, physical exam skills, Diagnosis, Differential
Diagnosis, patient care and management |
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The
objective of these WIN ROUNDS is to prepare our student
well to face these challenges. These WIN ROUNDS will
consist of Questions and Answers, Tables and Charts,
High Yield material and most commonly asked questions
on rounds in the hospital. This will help our students
to improve the performance and obtain better evaluations
from the attending physicians. THESE WIN ROUNDS ARE
AVAILABLE FOR OUR WINDSOR STUDENTS ONLY. PLEASE CONTACT
OUR OFFICE FOR THE PASSWORDS AND AUTHORIZATION. |
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SAMPLE of Question and Answer: |
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what is your preliminary diagnosis in a patient with
tachycardia tachypnea, increased P 2, S 3 and S 4
gallop, cracles, friction rub and a history of deep
venous thrombosis?
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Ans:
Pulmonary Thromboembolism |
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2.What
is your initial diagnosis in a patient with hyperresonance
and diminished breath sounds on one side of the chest?
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Ans:
Pneumothorax |
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SAMPLE of Table: |
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Test
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Finding
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Diagnostic possibilities
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Blood
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Hematocrit
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Increased
( > 55 percent )
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polycythemia
Secondary to chronic phypoxemia, e.g COPD, bronchitc
type
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WBCs
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hyperleukocytosis
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COPD
with superimposed in fection; bacterial pneumonia
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Eosinophilia
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Asthma
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Arterial
oxygen tension
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Decreased
(<75 mmHg)
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Hypoxemia
may be present in all pulmonary cuases of chronic
dyspnea.
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Arterial
carbon dioxide tension
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Increased
(>50 mmHg)
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hypercapnea:
in COPD, pre dominant emphysema; interstitial
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Alpha-1-
globulin
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Decreased
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alpah - 1 - antitrypsin deficiency.
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